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How does the visual cortex reorganize following complete agenesis of the corpus callosum, the largest commissure in the brain? If the widespread behavioral impairments that characterize autism are manifestations of poor functional connectivity, what is the nature of structural connectivity in autism? What alterations in the cortical areas that mediate face processing could give rise to a lifelong impairment in face recognition, as observed in congenital prosopagnosia (CP)?
Although these questions clearly revolve around disparate populations with neurodevelopmental disorders, the central theme of my research is to generate an understanding of the mechanisms underlying the development of the organization and function of the visual system by studying individuals in whom normal cortical development is perturbed. To this end, I use diffusion tensor tracking (DTT), functional MRI (fMRI) and behavioral techniques to examine the extent of alterations in structural and functional connectivity and its behavioral consequences.
Contact details:
Work: (412) 268-8117
Mail: cibu'at'cmu.edu
Thesis-Talk
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